Abstract Increasing evidence implicates altered mTORC1 signaling cascades within the pathophysiology of depression, suggesting that immediate modulation of mTORC1 signaling may offer novel therapeutic potential. In this problem with the JCI, Kato and colleagues documented that administration of NV-5138, a just lately produced synthetic leucine analog, features a swift and https://ezetimibe98654.frewwebs.com/30126567/the-bms-303141-diaries